eprintid: 1558227
rev_number: 32
eprint_status: archive
userid: 608
dir: disk0/01/55/82/27
datestamp: 2017-06-01 13:05:21
lastmod: 2021-12-06 00:43:37
status_changed: 2017-07-17 15:14:01
type: article
metadata_visibility: show
creators_name: Schwalbe, EC
creators_name: Lindsey, JC
creators_name: Nakjang, S
creators_name: Crosier, S
creators_name: Smith, AJ
creators_name: Hicks, D
creators_name: Rafiee, G
creators_name: Hill, RM
creators_name: Iliasova, A
creators_name: Stone, T
creators_name: Pizer, B
creators_name: Michalski, A
creators_name: Joshi, A
creators_name: Wharton, SB
creators_name: Jacques, TS
creators_name: Bailey, S
creators_name: Williamson, D
creators_name: Clifford, SC
title: Novel molecular subgroups for clinical classification and outcome prediction in childhood medulloblastoma: a cohort study
ispublished: pub
divisions: UCL
divisions: B02
divisions: D13
divisions: G22
keywords: Science & Technology, Life Sciences & Biomedicine, Oncology, BETA-CATENIN STATUS, CURRENT CONSENSUS, GENETIC PROFILES, TUMOR, INHIBITION, EXPRESSION, MUTATIONS, PATHWAY, GENOME
note: This work is licensed under a Creative Commons Attribution 4.0 International License. The images
or other third party material in this article are included in the article’s Creative Commons license,
unless indicated otherwise in the credit line; if the material is not included under the Creative Commons license,
users will need to obtain permission from the license holder to reproduce the material. To view a copy of this
license, visit http://creativecommons.org/licenses/by/4.0/
abstract: Background International consensus recognises four medulloblastoma molecular subgroups: WNT (MBWNT), SHH
(MBSHH), group 3 (MBGrp3), and group 4 (MBGrp4), each defined by their characteristic genome-wide transcriptomic and
DNA methylomic profiles. These subgroups have distinct clinicopathological and molecular features, and underpin
current disease subclassification and initial subgroup-directed therapies that are underway in clinical trials. However,
substantial biological heterogeneity and differences in survival are apparent within each subgroup, which remain to
be resolved. We aimed to investigate whether additional molecular subgroups exist within childhood medulloblastoma
and whether these could be used to improve disease subclassification and prognosis predictions.
Methods In this retrospective cohort study, we assessed 428 primary medulloblastoma samples collected from UK
Children’s Cancer and Leukaemia Group (CCLG) treatment centres (UK), collaborating European institutions, and
the UKCCSG-SIOP-PNET3 European clinical trial. An independent validation cohort (n=276) of archival tumour
samples was also analysed. We analysed samples from patients with childhood medulloblastoma who were aged
0–16 years at diagnosis, and had central review of pathology and comprehensive clinical data. We did comprehensive
molecular profiling, including DNA methylation microarray analysis, and did unsupervised class discovery of test and
validation cohorts to identify consensus primary molecular subgroups and characterise their clinical and biological
significance. We modelled survival of patients aged 3–16 years in patients (n=215) who had craniospinal irradiation
and had been treated with a curative intent.
Findings Seven robust and reproducible primary molecular subgroups of childhood medulloblastoma were identified.
MBWNT remained unchanged and each remaining consensus subgroup was split in two. MBSHH was split into agedependent
subgroups corresponding to infant (<4·3 years; MBSHH-Infant; n=65) and childhood patients (≥4·3 years;
MBSHH-Child; n=38). MBGrp3 and MBGrp4 were each split into high-risk (MBGrp3-HR [n=65] and MBGrp4-HR [n=85]) and low-risk
(MBGrp3-LR [n=50] and MBGrp4-LR [n=73]) subgroups. These biological subgroups were validated in the independent cohort.
We identified features of the seven subgroups that were predictive of outcome. Cross-validated subgroup-dependent
survival models, incorporating these novel subgroups along with secondary clinicopathological and molecular features
and established disease risk-factors, outperformed existing disease risk-stratification schemes. These subgroupdependent
models stratified patients into four clinical risk groups for 5-year progression-free survival: favourable risk
(54 [25%] of 215 patients; 91% survival [95% CI 82–100]); standard risk (50 [23%] patients; 81% survival [70–94]); highrisk
(82 [38%] patients; 42% survival [31–56]); and very high-risk (29 [13%] patients; 28% survival [14–56]).
Interpretation The discovery of seven novel, clinically significant subgroups improves disease risk-stratification and
could inform treatment decisions. These data provide a new foundation for future research and clinical investigations.
date: 2017-05-22
date_type: published
publisher: ELSEVIER SCIENCE INC
official_url: http://doi.org/10.1016/S1470-2045(17)30243-7
oa_status: green
full_text_type: pub
language: eng
primo: open
primo_central: open_green
article_type_text: Article
verified: verified_manual
elements_id: 1296612
doi: 10.1016/S1470-2045(17)30243-7
lyricists_name: Jacques, Thomas
lyricists_name: Stone, Thomas
lyricists_id: TJACQ32
lyricists_id: STONE38
actors_name: Jacques, Thomas
actors_name: Stacey, Thomas
actors_id: TJACQ32
actors_id: TSSTA20
actors_role: owner
actors_role: impersonator
full_text_status: public
publication: Lancet Oncology
volume: 18
number: 7
pagerange: 958-971
pages: 14
issn: 1474-5488
citation:        Schwalbe, EC;    Lindsey, JC;    Nakjang, S;    Crosier, S;    Smith, AJ;    Hicks, D;    Rafiee, G;                                             ... Clifford, SC; + view all <#>        Schwalbe, EC;  Lindsey, JC;  Nakjang, S;  Crosier, S;  Smith, AJ;  Hicks, D;  Rafiee, G;  Hill, RM;  Iliasova, A;  Stone, T;  Pizer, B;  Michalski, A;  Joshi, A;  Wharton, SB;  Jacques, TS;  Bailey, S;  Williamson, D;  Clifford, SC;   - view fewer <#>    (2017)    Novel molecular subgroups for clinical classification and outcome prediction in childhood medulloblastoma: a cohort study.                   Lancet Oncology , 18  (7)   pp. 958-971.    10.1016/S1470-2045(17)30243-7 <https://doi.org/10.1016/S1470-2045%2817%2930243-7>.       Green open access   
 
document_url: https://discovery.ucl.ac.uk/id/eprint/1558227/1/Jacques-T_novel%20molecular_classification_child_.pdf