eprintid: 1544814
rev_number: 35
eprint_status: archive
userid: 608
dir: disk0/01/54/48/14
datestamp: 2017-03-12 00:37:33
lastmod: 2021-10-06 22:38:33
status_changed: 2017-11-28 16:50:44
type: article
metadata_visibility: show
creators_name: Gandhi, S
creators_name: Plun-Favreau, H
title: Mutations and mechanism: how PINK1 may contribute to risk of sporadic Parkinson's disease
ispublished: pub
divisions: UCL
divisions: B02
divisions: C07
divisions: D07
divisions: F84
divisions: F86
keywords: Science & Technology, Life Sciences & Biomedicine, Clinical Neurology, Neurosciences, Neurosciences & Neurology, Mitophagy
note: This version is the author accepted manuscript. For information on re-use, please refer to the publisher’s terms and conditions.
abstract: This scientific commentary refers to
‘Heterozygous PINK1 p.G411S increases
risk of Parkinson’s disease
via a dominant-negative mechanism’,
by Puschmann et al. (doi:10.1093/
brain/aww261).
date: 2017-01-01
date_type: published
publisher: OXFORD UNIV PRESS
official_url: http://doi.org/10.1093/brain/aww320
oa_status: green
language: eng
primo: open
primo_central: open_green
article_type_text: Editorial Material
verified: verified_manual
elements_id: 1199279
doi: 10.1093/brain/aww320
lyricists_name: Gandhi, Sonia
lyricists_name: Plun-Favreau, Helene
lyricists_id: SGAND43
lyricists_id: HPLUN15
full_text_status: public
publication: Brain
volume: 140
number: 1
pagerange: 2-5
pages: 4
issn: 1460-2156
citation:        Gandhi, S;    Plun-Favreau, H;      (2017)    Mutations and mechanism: how PINK1 may contribute to risk of sporadic Parkinson's disease.                   Brain , 140  (1)   pp. 2-5.    10.1093/brain/aww320 <https://doi.org/10.1093/brain%2Faww320>.       Green open access   
 
document_url: https://discovery.ucl.ac.uk/id/eprint/1544814/1/Plun-Favreau_BRAIN_Puschmann_commentary.pdf