@article{discovery1472457,
            year = {2015},
          volume = {16},
           month = {June},
         journal = {BMC Bioinformatics},
           title = {Using Gene Ontology to describe the role of the neurexin-neuroligin-SHANK complex in human, mouse and rat and its relevance to autism},
            note = {{\copyright} 2015 Patel et al.
This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.},
            issn = {1471-2105},
        keywords = {Animals, Autistic Disorder, Cell Adhesion Molecules, Neuronal, Cell Physiological Phenomena, Gene Ontology, Genome, Genomics, High-Throughput Nucleotide Sequencing, Humans, Membrane Potentials, Mice, Models, Molecular, Nerve Tissue Proteins, Phenotype, Rats, Social Behavior, Synapses, Synaptic Potentials},
        abstract = {People with an autistic spectrum disorder (ASD) display a variety of characteristic behavioral traits, including impaired social interaction, communication difficulties and repetitive behavior. This complex neurodevelopment disorder is known to be associated with a combination of genetic and environmental factors. Neurexins and neuroligins play a key role in synaptogenesis and neurexin-neuroligin adhesion is one of several processes that have been implicated in autism spectrum disorders.},
          author = {Patel, S and Roncaglia, P and Lovering, RC},
             url = {http://dx.doi.org/10.1186/s12859-015-0622-0}
}