%0 Thesis %9 Doctoral %A Pawlik, Piotr %B UCL Cancer Institute %D 2025 %F discovery:10203239 %I UCL (University College London) %P 207 %T Inference of clone-specific copy numbers from bulk tumour DNA %U https://discovery.ucl.ac.uk/id/eprint/10203239/ %X Cancer is an evolutionary process. During this process, single nucleotide variants (SNVs) and somatic copy-number alterations (SCNAs) accumulate, contributing to clonal evolution. Nevertheless, making accurate inferences about their co-evolution and clone-specific copy numbers from bulk DNA sequencing poses a significant challenge. In this thesis, I present ALPACA (ALlelespecific Phylogenetic Analysis of clone Copy-number Alterations), a computational method which allows inference of clone-specific copy-numbers from bulk DNA by leveraging phylogenetic trees and SNV cellular frequencies obtained from multi-sample sequencing experiments. I validate this approach with two simulated tumour cohorts and with single-cell data, comparing the ALPACA output both with the ground truth and with the outputs of two previously published methods. I then apply ALPACA to the TRACERx421 (TRAcking non-small cell lung Cancer Evolution through therapy Frankell et al. 2023; Al Bakir et al. 2023) cohort and demonstrate that ALPACA can identify loss-ofheterozygosity events and amplifications in minor metastasis seeding clones, not detectable on bulk-sample copy-number level. By comparing seeding and non-seeding clones within the primary-metastatic matched cohort I observe evidence of increased chromosomal instability in metastasis seeding clones. Using ALPACA’s clone-specific copy-numbers I develop a novel method to quantify intra-tumour copy-number heterogeneity: clone copy-number diversity (CCD). Using this metric I show that patients who later develop a metastatic disease harbour a higher intra-tumour clone copy number diversity within their primary tumours compared to patients who do not relapse. Lastly, I show an association between CCD and patient survival. %Z Copyright © The Author 2025. Original content in this thesis is licensed under the terms of the Creative Commons Attribution-NonCommercial 4.0 International (CC BY-NC 4.0) Licence (https://creativecommons.org/licenses/by-nc/4.0/). Any third-party copyright material present remains the property of its respective owner(s) and is licensed under its existing terms. Access may initially be restricted at the author’s request.