TY - JOUR TI - The role of complement in kidney disease: conclusions from a Kidney Disease: Improving Global Outcomes (KDIGO) Controversies Conference AV - public Y1 - 2024/09// EP - 391 N2 - Uncontrolled complement activation can cause or contribute to glomerular injury in multiple kidney diseases. Although complement activation plays a causal role in atypical hemolytic uremic syndrome and C3 glomerulopathy, over the past decade, a rapidly accumulating body of evidence has shown a role for complement activation in multiple other kidney diseases, including diabetic nephropathy and several glomerulonephritides. The number of available complement inhibitor therapies has also increased during the same period. In 2022, Kidney Diseases: Improving Global Outcomes (KDIGO) convened a Controversies Conference, ?The Role of Complement in Kidney Disease,? to address the expanding role of complement dysregulation in the pathophysiology, diagnosis, and management of various glomerular diseases, diabetic nephropathy, and other forms of hemolytic uremic syndrome. Conference participants reviewed the evidence for complement playing a primary causal or secondary role in progression for several disease states and considered how evidence of complement involvement might inform management. Participating patients with various complement-mediated diseases and caregivers described concerns related to life planning, implications surrounding genetic testing, and the need for inclusive implementation of effective novel therapies into clinical practice. The value of biomarkers in monitoring disease course and the role of the glomerular microenvironment in complement response were examined, and key gaps in knowledge and research priorities were identified. ID - discovery10196186 PB - Elsevier BV KW - Complement inhibitor; complement-mediated injury; glomerular injury VL - 106 SP - 369 IS - 3 N1 - Copyright © 2024 Kidney Disease: Improving Global Outcomes (KDIGO). Published by Elsevier Inc. on behalf of the International Society of Nephrology. This is an open access article under the CC BY-NC-ND license (http:// creativecommons.org/licenses/by-nc-nd/4.0/). UR - http://dx.doi.org/10.1016/j.kint.2024.05.015 SN - 0085-2538 JF - Kidney International A1 - Vivarelli, Marina A1 - Barratt, Jonathan A1 - Beck Jr, Laurence H A1 - Fakhouri, Fadi A1 - Gale, Daniel P A1 - Goicoechea de Jorge, Elena A1 - Mosca, Marta A1 - Noris, Marina A1 - Pickering, Matthew C A1 - Susztak, Katalin A1 - Thurman, Joshua M A1 - Cheung, Michael A1 - King, Jennifer M A1 - Jadoul, Michel A1 - Winkelmayer, Wolfgang C A1 - Smith, Richard JH ER -