TY - JOUR TI - Development and translation of thiometallate sulfide donors using a porcine model of coronary occlusion and reperfusion Y1 - 2024/07// AV - public VL - 73 N1 - Crown Copyright © 2024 Published by Elsevier B.V. This is an open access article under the CC BY-NC license (http://creativecommons.org/licenses/bync/4.0/). N2 - Sulfide-releasing compounds reduce reperfusion injury by decreasing mitochondria-derived reactive oxygen species production. We previously characterised ammonium tetrathiomolybdate (ATTM), a clinically used copper chelator, as a sulfide donor in rodents. Here we assessed translation to large mammals prior to clinical testing. In healthy pigs an intravenous ATTM dose escalation revealed a reproducible pharmacokinetic/pharmacodynamic (PK/PD) relationship with minimal adverse clinical or biochemical events. In a myocardial infarction (1-h occlusion of the left anterior descending coronary artery)-reperfusion model, intravenous ATTM or saline was commenced just prior to reperfusion. ATTM protected the heart (24-h histological examination) in a drug-exposure-dependent manner (r2 = 0.58, p < 0.05). Blood troponin T levels were significantly (p < 0.05) lower in ATTM-treated animals while myocardial glutathione peroxidase activity, an antioxidant selenoprotein, was elevated (p < 0.05). Overall, our study represents a significant advance in the development of sulfides as therapeutics and underlines the potential of ATTM as a novel adjunct therapy for reperfusion injury. Mechanistically, our study suggests that modulating selenoprotein activity could represent an additional mode of action of sulfide-releasing drugs. ID - discovery10191937 UR - http://dx.doi.org/10.1016/j.redox.2024.103167 PB - Elsevier BV SN - 2213-2317 JF - Redox Biology A1 - Johnson, Thomas W A1 - Holt, James A1 - Kleyman, Anna A1 - Zhou, Shengyu A1 - Sammut, Eva A1 - Bruno, Vito Domenico A1 - Gaupp, Charlotte A1 - Stanzani, Giacomo A1 - Martin, John A1 - Arina, Pietro A1 - Deutsch, Julia A1 - Ascione, Raimondo A1 - Singer, Mervyn A1 - Dyson, Alex KW - Mitochondria; Ischaemia; Reperfusion injury; Reactive oxygen species; Selenoprotein; Tetrathiomolybdate; Gasotransmitter ER -