@article{discovery10131989,
            note = {This version is the author accepted manuscript. For information on re-use, please refer to the publisher's terms and conditions.},
       publisher = {Springer Science and Business Media LLC},
           month = {June},
           pages = {498--499},
         journal = {Nature Aging},
          volume = {1},
            year = {2021},
           title = {The phosphorylation cascade hypothesis of Alzheimer's disease},
          number = {6},
        abstract = {Alzheimer's disease (AD) is characterized by amyloid-{\ensuremath{\beta}} (A{\ensuremath{\beta}})-induced phosphorylation of the axon-stabilizing tau protein, which causes neurodegeneration. Here, Morshed et al. show that deregulated phosphorylation in AD also affects other proteins and cell types in the brain, suggesting that the tau-centric view on A{\ensuremath{\beta}} toxicity should be revised.},
             url = {http://dx.doi.org/10.1038/s43587-021-00077-9},
          author = {Brinkmalm, G and Zetterberg, H}
}