TY - UNPB N1 - Thesis digitised by ProQuest. PB - UCL (University College London) A1 - Teo, Mabel AV - public TI - Purification and functional characterization of brain and recombinant chimaerin, a p21rac GTPase activating protein M1 - Doctoral Y1 - 1994/// UR - https://discovery.ucl.ac.uk/id/eprint/10098872/ ID - discovery10098872 N2 - n-chimaerin is a GAP for the ras-related p21rac. A 45 kDa brain protein (p45) immune reactive to anti-n-chimaerin polyclonal antibodies had selective p21 rac GAP activity, using an overlay assay. p45-chimaerin was purified 400- fold from rat brain by column chromatography. Tryptic peptides contained sequences identical to that predicted from ?2-chimaerin cDNA, a splice variant encoding a divergent N-terminal with a SH2 domain. Thus, p45-chimaerin probably corresponds to SH2-containing (?2) chimaerin. A 35 kDa p21rac GAP (p35) detected in detergent soluble membrane fractions, immunoreactive to chimaerin antiserum was likely to represent n-chimaerin (?1-chimaerin). Diverse GAPs for the rho/rac family were present in the brain; p45-chimaerin was widely distributed in brain regions except cerebellum, and was present in both membrane and cytosolic fractions. Both native and recombinant a2-chimaerin exhibited p21rac GAP activity in solution, which was stimulated by phosphatidylserine with a synergistic effect by phorbol esters. GAP activity of ?2-chimaerin was unaffected by its SH2 domain. In contrast to ?1-chimaerin, ?2-chimaerin bound more phorbol ester in the presence of phosphatidylinositol than of phosphatidylserine. ?2-Chimaerin was phosphorylated by PKC and PKA in vitro. Brain proteins interacting with ?2-chimaerin were detected using 32P-labelled PKC phosphorylated chimaerin. A 60 kDa protein interacting with the SH2 domain of ?2-chimaerin was purified; peptide sequences showed it to be novel. Two peptides had similarity to the consensus sequences of a MAP kinase substrate and a SH2 binding domain. These data suggest that ?2-chimaerin plays a physiological role in neuronal signal transduction involving SH2-linked receptor/tyrosine kinase and p21rac signalling pathways. ER -