<> <http://www.w3.org/2000/01/rdf-schema#comment> "The repository administrator has not yet configured an RDF license."^^<http://www.w3.org/2001/XMLSchema#string> .
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<https://discovery.ucl.ac.uk/id/eprint/10065062> <http://purl.org/dc/terms/title> "Transient increase in CSF GAP-43 concentration after ischemic stroke"^^<http://www.w3.org/2001/XMLSchema#string> .
<https://discovery.ucl.ac.uk/id/eprint/10065062> <http://purl.org/ontology/bibo/abstract> "Background: Cerebrospinal fluid (CSF) biomarkers reflect ongoing processes in the brain. Growth-associated\r\nprotein 43 (GAP-43) is highly upregulated in brain tissue shortly after experimental ischemia suggesting the CSF\r\nGAP-43 concentration may be altered in ischemic brain disorders. CSF GAP-43 concentration is elevated in\r\nAlzheimer’s disease patients; however, patients suffering from stroke have not been studied previously.\r\nMethods: The concentration of GAP-43 was measured in longitudinal CSF samples from 28 stroke patients\r\nprospectively collected on days 0–1, 2–4, 7–9, 3 weeks, and 3–5 months after ischemia and cross-sectionally in 19\r\ncontrols. The stroke patients were clinically evaluated using a stroke severity score system. The extent of the brain\r\nlesion, including injury size and degrees of white matter lesions and atrophy were evaluated by CT and magnetic\r\nresonance imaging.\r\nResults: Increased GAP-43 concentration was detected from day 7–9 to 3 weeks after stroke, compared to day 1–4\r\nand to levels in the control group (P = 0.02 and P = 0.007). At 3–5 months after stroke GAP-43 returned to admission\r\nlevels. The initial increase in GAP-43 during the nine first days was associated to stroke severity, the degree of white\r\nmatter lesions and atrophy and correlated positively with infarct size (rs = 0.65, P = 0.001).\r\nConclusions: The transient increase of CSF GAP-43 is important to take into account when used as a biomarker for\r\nother neurodegenerative diseases such as Alzheimer’s disease. Furthermore, GAP-43 may be a marker of neuronal\r\nresponses after stroke and additional studies confirming the potential of CSF GAP-43 to reflect severity and\r\noutcome of stroke in larger cohorts are warranted."^^<http://www.w3.org/2001/XMLSchema#string> .
<https://discovery.ucl.ac.uk/id/eprint/10065062> <http://purl.org/dc/terms/date> "2018-12-07" .
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