%D 2017
%K Science & Technology, Life Sciences & Biomedicine, Clinical Neurology, Psychiatry, Surgery, Neurosciences & Neurology, ACUTE ISCHEMIC-STROKE, DEPRESSION FOLLOWING STROKE, PRIMARY MOTOR CORTEX, CEREBRAL-ISCHEMIA, BASAL GANGLIA, ACUTE-PHASE, FOLLOW-UP, INDOLEAMINE 2,3-DIOXYGENASE, PROINFLAMMATORY CYTOKINES, PSYCHOMETRIC QUALITIES
%T Mechanisms of poststroke fatigue
%N 3
%P 287-293
%V 89
%J Journal of Neurology, Neurosurgery & Psychiatry
%A W De Doncker
%A R Dantzer
%A H Ormstad
%A A Kuppuswamy
%O This version is the author accepted manuscript. For information on re-use, please refer to the publisher’s terms and conditions.
%I BMJ PUBLISHING GROUP
%L discovery10059401
%X Poststroke fatigue is a debilitating symptom and is poorly understood. Here we summarise molecular, behavioural and neurophysiological changes related to poststroke fatigue and put forward potential theories for mechanistic understanding of poststroke fatigue.