%D 2017 %K Science & Technology, Life Sciences & Biomedicine, Clinical Neurology, Psychiatry, Surgery, Neurosciences & Neurology, ACUTE ISCHEMIC-STROKE, DEPRESSION FOLLOWING STROKE, PRIMARY MOTOR CORTEX, CEREBRAL-ISCHEMIA, BASAL GANGLIA, ACUTE-PHASE, FOLLOW-UP, INDOLEAMINE 2,3-DIOXYGENASE, PROINFLAMMATORY CYTOKINES, PSYCHOMETRIC QUALITIES %T Mechanisms of poststroke fatigue %N 3 %P 287-293 %V 89 %J Journal of Neurology, Neurosurgery & Psychiatry %A W De Doncker %A R Dantzer %A H Ormstad %A A Kuppuswamy %O This version is the author accepted manuscript. For information on re-use, please refer to the publisher’s terms and conditions. %I BMJ PUBLISHING GROUP %L discovery10059401 %X Poststroke fatigue is a debilitating symptom and is poorly understood. Here we summarise molecular, behavioural and neurophysiological changes related to poststroke fatigue and put forward potential theories for mechanistic understanding of poststroke fatigue.