TY - JOUR VL - 18 IS - 10 N1 - This article is distributed under the terms of the Creative Commons Attribution 4.0 International License (http:// creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution, and reproduction in any medium, provided you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. UR - http://doi.org/10.1007/s11910-018-0874-y SN - 1534-6293 A1 - Orme, T A1 - Guerreiro, R A1 - Bras, J JF - Current Neurology and Neuroscience Reports Y1 - 2018/10// AV - public EP - 13 TI - The Genetics of Dementia with Lewy Bodies: Current Understanding and Future Directions PB - SPRINGER N2 - PURPOSE OF REVIEW: Dementia with Lewy bodies (DLB) is a neurodegenerative disease that can be clinically and pathologically similar to Parkinson?s disease (PD) and Alzheimer?s disease (AD). Current understanding of DLB genetics is insufficient and has been limited by sample size and difficulty in diagnosis. The first genome-wide association study (GWAS) in DLB was performed in 2017; a time at which the post-GWAS era has been reached in many diseases. RECENT FINDINGS: DLB shares risk loci with AD, in the APOE E4 allele, and with PD, in variation at GBA and SNCA. Interestingly, the GWAS suggested that DLB may also have genetic risk factors that are distinct from those in AD and PD. SUMMARY: Although off to a slow start, recent studies have reinvigorated the field of DLB genetics and these results enable us to start to have a more complete understanding of the genetic architecture of this disease. ID - discovery10055017 KW - Science & Technology KW - Life Sciences & Biomedicine KW - Clinical Neurology KW - Neurosciences KW - Neurosciences & Neurology KW - Dementia with Lewy bodies KW - DLB KW - Genetics KW - GWAS KW - Next-generation sequencing KW - Genome-Wide Association KW - Alpha-Synuclein Gene KW - Familial Parkinsons-Disease KW - Beta-Glucocerebrosidase Activity KW - Alzheimers-Disease KW - Apolipoprotein-E KW - Body Disease KW - Frontotemporal Dementia KW - Locus Triplication KW - Gaucher-Disease ER -