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<https://discovery.ucl.ac.uk/id/eprint/10048831> <http://purl.org/ontology/bibo/abstract> "Болезнь Паркинсона (БП) - одно из самых распространенных нейродегенеративных заболеваний. Развитие патологии связано с гибелью дофаминергических нейронов, главным образом, в черной субстанции головного мозга. Недостаточность дофамина вызывает целый набор тяжелых симптомов, среди которых брадикинезия, постуральная неустойчивость, ригидность мышц и тремор. Генетические исследования показали, что ведущую роль в патогенезе БП играет белок альфа-синуклеин (?-Син). Большое количество данных свидетельствует о различных механизмах токсического действия ?-Син. Кроме того, среди ключевых факторов, способствующих развитию нейродегенерации при БП, выделяют существенные нарушения функций митохондрий и/или мутации. В число мутируемых генов при наследственной и спорадической формах БП входят гены, кодирующие PINK1 и Parkin, основные компоненты системы “контроля качества” митохондрий. Самые ранние биохимические признаки заболевания проявляются в нарушениях взаимодействия митохондрий и эндоплазматического ретикулума, митохондриальной динамики, гомеостаза кальция и повышении уровня митохондриальных активных форм кислорода. Все эти факторы участвуют в повреждении дофаминергических нейронов."^^<http://www.w3.org/2001/XMLSchema#string> .
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