eprintid: 10047119
rev_number: 36
eprint_status: archive
userid: 608
dir: disk0/10/04/71/19
datestamp: 2018-04-19 14:45:17
lastmod: 2021-09-20 00:11:39
status_changed: 2018-04-19 14:45:17
type: article
metadata_visibility: show
creators_name: Taskesen, E
creators_name: Mishra, A
creators_name: Van Der Sluis, S
creators_name: Ferrari, R
creators_name: Veldink, JH
creators_name: Van Es, MA
creators_name: Smit, AB
creators_name: Posthuma, D
creators_name: Pijnenburg, Y
title: Susceptible genes and disease mechanisms identified in frontotemporal dementia and frontotemporal dementia with Amyotrophic Lateral Sclerosis by DNA-methylation and GWAS
ispublished: pub
divisions: UCL
divisions: B02
divisions: C07
divisions: DF9
divisions: FA5
divisions: D07
divisions: F84
divisions: F86
note: Copyright © The Author(s) 2017. Open Access: This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative
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abstract: Frontotemporal dementia (FTD) is a neurodegenerative disorder predominantly affecting the frontal and temporal lobes. Genome-wide association studies (GWAS) on FTD identified only a few risk loci. One of the possible explanations is that FTD is clinically, pathologically, and genetically heterogeneous. An important open question is to what extent epigenetic factors contribute to FTD and whether these factors vary between FTD clinical subgroup. We compared the DNA-methylation levels of FTD cases (n = 128), and of FTD cases with Amyotrophic Lateral Sclerosis (FTD-ALS; n = 7) to those of unaffected controls (n = 193), which resulted in 14 and 224 candidate genes, respectively. Cluster analysis revealed significant class separation of FTD-ALS from controls. We could further specify genes with increased susceptibility for abnormal gene-transcript behavior by jointly analyzing DNA-methylation levels with the presence of mutations in a GWAS FTD-cohort. For FTD-ALS, this resulted in 9 potential candidate genes, whereas for FTD we detected 1 candidate gene (ELP2). Independent validation-sets confirmed the genes DLG1, METTL7A, KIAA1147, IGHMBP2, PCNX, UBTD2, WDR35, and ELP2/SLC39A6 among others. We could furthermore demonstrate that genes harboring mutations and/or displaying differential DNA-methylation, are involved in common pathways, and may therefore be critical for neurodegeneration in both FTD and FTD-ALS.
date: 2017-12-01
date_type: published
official_url: http://dx.doi.org/10.1038/s41598-017-09320-z
oa_status: green
full_text_type: pub
language: eng
primo: open
primo_central: open_green
article_type_text: Journal Article
verified: verified_manual
elements_id: 1549470
doi: 10.1038/s41598-017-09320-z
lyricists_name: Collinge, John
lyricists_name: Ferrari, Raffaele
lyricists_name: Fox, Nicholas
lyricists_name: Hardy, John
lyricists_name: Mead, Simon
lyricists_name: Morris, Huw
lyricists_name: Rossor, Martin
lyricists_name: Smalley, June
lyricists_name: Warren, Jason
lyricists_id: JCOLL20
lyricists_id: RFERR12
lyricists_id: NCIFO25
lyricists_id: JHARD28
lyricists_id: SMEAD68
lyricists_id: HRMOR79
lyricists_id: MNROS52
lyricists_id: JASMA87
lyricists_id: JDWAR75
actors_name: Bracey, Alan
actors_id: ABBRA90
actors_role: owner
full_text_status: public
publication: Scientific Reports
volume: 7
article_number: 8899
issn: 2045-2322
citation:        Taskesen, E;    Mishra, A;    Van Der Sluis, S;    Ferrari, R;    Veldink, JH;    Van Es, MA;    Smit, AB;         ... Pijnenburg, Y; + view all <#>        Taskesen, E;  Mishra, A;  Van Der Sluis, S;  Ferrari, R;  Veldink, JH;  Van Es, MA;  Smit, AB;  Posthuma, D;  Pijnenburg, Y;   - view fewer <#>    (2017)    Susceptible genes and disease mechanisms identified in frontotemporal dementia and frontotemporal dementia with Amyotrophic Lateral Sclerosis by DNA-methylation and GWAS.                   Scientific Reports , 7     , Article 8899.  10.1038/s41598-017-09320-z <https://doi.org/10.1038/s41598-017-09320-z>.       Green open access   
 
document_url: https://discovery.ucl.ac.uk/id/eprint/10047119/1/s41598-017-09320-z.pdf
document_url: https://discovery.ucl.ac.uk/id/eprint/10047119/8/Taskesen_Susceptible_genes_disease_mechanisms_Suppl.pdf