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A zebrafish model of CLN2 disease is deficient in tripeptidyl peptidase 1 and displays progressive neurodegeneration accompanied by a reduction in proliferation

Mahmood, F; Fu, S; Cooke, J; Wilson, SW; Cooper, JD; Russell, C; (2013) A zebrafish model of CLN2 disease is deficient in tripeptidyl peptidase 1 and displays progressive neurodegeneration accompanied by a reduction in proliferation. BRAIN , 136 pp. 1488-1507. 10.1093/brain/awt043.

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Type: Article
Title: A zebrafish model of CLN2 disease is deficient in tripeptidyl peptidase 1 and displays progressive neurodegeneration accompanied by a reduction in proliferation
DOI: 10.1093/brain/awt043
Keywords: Science & Technology, Life Sciences & Biomedicine, Clinical Neurology, Neurosciences, Neurosciences & Neurology, tripeptidyl peptidase 1, TPP1, CLN2 disease, late infantile neuronal ceroid lipofuscinosis, lysosomal storage disorder, zebrafish, model, NEURONAL CEROID-LIPOFUSCINOSIS, MITOCHONDRIAL ATP SYNTHASE, LATE-INFANTILE, SUBUNIT-C, MOUSE MODEL, IN-VIVO, HYDROPHOBIC PROTEIN, LARVAL ZEBRAFISH, CELL-DEATH, JUVENILE
UCL classification: UCL > Provost and Vice Provost Offices
UCL > Provost and Vice Provost Offices > School of Life and Medical Sciences
UCL > Provost and Vice Provost Offices > School of Life and Medical Sciences > Faculty of Life Sciences
UCL > Provost and Vice Provost Offices > School of Life and Medical Sciences > Faculty of Life Sciences > Div of Biosciences
UCL > Provost and Vice Provost Offices > School of Life and Medical Sciences > Faculty of Life Sciences > Div of Biosciences > Cell and Developmental Biology
URI: http://discovery.ucl.ac.uk/id/eprint/1398784
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