Lyons, PA; Rayner, TF; Trivedi, S; Holle, JU; Watts, RA; Jayne, DR; ... Smith, KG; + view all Lyons, PA; Rayner, TF; Trivedi, S; Holle, JU; Watts, RA; Jayne, DR; Baslund, B; Brenchley, P; Bruchfeld, A; Chaudhry, AN; Cohen Tervaert, JW; Deloukas, P; Feighery, C; Gross, WL; Guillevin, L; Gunnarsson, I; Harper, L; Hrušková, Z; Little, MA; Martorana, D; Neumann, T; Ohlsson, S; Padmanabhan, S; Pusey, CD; Salama, AD; Sanders, JS; Savage, CO; Segelmark, M; Stegeman, CA; Tesař, V; Vaglio, A; Wieczorek, S; Wilde, B; Zwerina, J; Rees, AJ; Clayton, DG; Smith, KG; - view fewer (2012) Genetically distinct subsets within ANCA-associated vasculitis. N Engl J Med , 367 (3) 214 - 223. 10.1056/NEJMoa1108735.
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Antineutrophil cytoplasmic antibody (ANCA)-associated vasculitis is a severe condition encompassing two major syndromes: granulomatosis with polyangiitis (formerly known as Wegener's granulomatosis) and microscopic polyangiitis. Its cause is unknown, and there is debate about whether it is a single disease entity and what role ANCA plays in its pathogenesis. We investigated its genetic basis.
|Title:||Genetically distinct subsets within ANCA-associated vasculitis.|
|Additional information:||PMCID: PMC3773907|
|Keywords:||Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis, Case-Control Studies, Female, Genetic Predisposition to Disease, Genome-Wide Association Study, Genotyping Techniques, HLA-DP Antigens, Humans, Major Histocompatibility Complex, Male, Microscopic Polyangiitis, Myeloblastin, Polymorphism, Single Nucleotide, Risk Factors, Wegener Granulomatosis, alpha 1-Antitrypsin|
|UCL classification:||UCL > School of Life and Medical Sciences > Faculty of Medical Sciences > Medicine (Division of) > Internal Medicine|
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